Renal infarction

CT

First and second year — the floor first, then every step

Flank pain with no stone: a wedge-shaped, non-enhancing cortical defect with the cortical rim sign, or global non-enhancement — then the cause (embolus from the heart, dissection, thrombosis).

Orient first

  • Renal infarction is underdiagnosed because it mimics colic and pyelonephritis.
  • The cortical rim sign — a thin enhancing subcapsular rim from capsular collaterals — separates infarct from pyelonephritis (after several hours).
  • Causes: cardioembolism (atrial fibrillation), renal artery dissection, aortic dissection, vasculitis, thrombophilia.

Acquire the study

  • CT with IV contrast: arterial and nephrographic phases; CT angiography when a renal artery or aortic cause is sought.

The manoeuvre

  • Nephrographic phase: sharply demarcated wedge-shaped non-enhancing area with its base at the capsule.
  • Cortical rim sign: a thin enhancing peripheral rim over the defect.
  • Arterial phase: renal artery or branch occlusion, dissection flap, aortic source.
  • Other organs: splenic infarcts, bowel ischaemia (embolic shower).
  • Look for cardiac thrombus if the heart is in the field.

What confirms it

  • A wedge-shaped non-enhancing renal defect with a rim sign or a demonstrated arterial occlusion.

What licenses you to exclude it

  • Homogeneous nephrographic enhancement of both kidneys excludes established infarction.

The classic misread

  • Calling a striated nephrogram of pyelonephritis an infarct — pyelonephritis is striated and has no rim sign.

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